SMARCD1 subunit of SWI SNF chromatin remodeling complexes collaborates with p53 to exert an oncogenic role in B ALL by maintaining high metabolic activity
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SMARCD1 subunit of SWI SNF chromatin remodeling complexes collaborates with p53 to exert an oncogenic role in B ALL by maintaining high metabolic activity
Alternating cycles of quiescent and proliferative cell states determine stemness and leukemia initiation capacity in acute lymphoblastic leukemia
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Alternating cycles of quiescent and proliferative cell states determine stemness and leukemia initiation capacity in acute lymphoblastic leukemia
SLC7A11 suppresses ferroptosis via arginine metabolism and promotes tumor immune escape in acute lymphoblastic leukemia
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SLC7A11 suppresses ferroptosis via arginine metabolism and promotes tumor immune escape in acute lymphoblastic leukemia
Targeting VDAC1 to induce mitochondrial DNA–STING signaling and lysosomal cell death in T cell lymphoma
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Targeting VDAC1 to induce mitochondrial DNA–STING signaling and lysosomal cell death in T cell lymphoma
EZH2 oncogenic mutation disrupts germinal center B cell dynamics and promotes early lymphomagenic behaviors
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EZH2 oncogenic mutation disrupts germinal center B cell dynamics and promotes early lymphomagenic behaviors
Murine modeling of cluster 1 diffuse large B cell lymphoma reveals transformation from marginal zone lymphoma
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Murine modeling of cluster 1 diffuse large B cell lymphoma reveals transformation from marginal zone lymphoma
The chromatin remodeler hells regulates transcription replication conflicts and genome integrity in ALK negative anaplastic large cell lymphoma
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The chromatin remodeler hells regulates transcription replication conflicts and genome integrity in ALK negative anaplastic large cell lymphoma
MYC dependent permissiveness of PI3K hyperactivation renders metabolic vulnerability in R R b ALL
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MYC dependent permissiveness of PI3K hyperactivation renders metabolic vulnerability in R R b ALL
Novel therapeutics for SF3B1 mutant cancers which exploit the missplicing of DCAF16
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Novel therapeutics for SF3B1 mutant cancers which exploit the missplicing of DCAF16