636. Myelodysplastic Syndromes: Basic and Translational: Poster II
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Interim Results of the CMML scrna seq atlas GMP skewing is independently associated with overall survival in CMML
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Interim Results of the CMML scrna seq atlas GMP skewing is independently associated with overall survival in CMML
Synergistic apoptotic effect by the combination of clk inhibitor CTX 712 and BCL 2 inhibitor ABT 199 with or without azacytidine in AML and MDS
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Synergistic apoptotic effect by the combination of clk inhibitor CTX 712 and BCL 2 inhibitor ABT 199 with or without azacytidine in AML and MDS
Treatment with luspatersept exerts a pleiotropic effect on the molecular signatures of bone marrow cell subsets
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Treatment with luspatersept exerts a pleiotropic effect on the molecular signatures of bone marrow cell subsets
Functional classification of monoallelic TP53 mutations refines prognostic stratification in myelodysplastic neoplasms
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Functional classification of monoallelic TP53 mutations refines prognostic stratification in myelodysplastic neoplasms
Integrated analysis of autoantibody profiles and clonal distribution of RAS mutations in juvenile myelomonocytic leukemia
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Integrated analysis of autoantibody profiles and clonal distribution of RAS mutations in juvenile myelomonocytic leukemia
Immunosuppressive T cell landscape in myelodysplastic Neoplasms with TP53 mutations
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Immunosuppressive T cell landscape in myelodysplastic Neoplasms with TP53 mutations
Spatial multiomics reveal a critical role for TIM4 expressing macrophages in blast transformation of chronic myelomonocytic leukemia
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Spatial multiomics reveal a critical role for TIM4 expressing macrophages in blast transformation of chronic myelomonocytic leukemia
Single cell multi omics profiling reveals distinct patterns of clonal expansion and microenvironmental shifts associated with disease evolution in patients with Myelodysplastic Syndromes
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Single cell multi omics profiling reveals distinct patterns of clonal expansion and microenvironmental shifts associated with disease evolution in patients with Myelodysplastic Syndromes
Examining the role of U2AF1 and TET2 mutations in a myeloid malignancy mouse model
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Examining the role of U2AF1 and TET2 mutations in a myeloid malignancy mouse model
Novel approaches to identify cellular vulnerabilities in TET2 dependent MDS
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Novel approaches to identify cellular vulnerabilities in TET2 dependent MDS