602. Myeloid Oncogenesis: Basic: Poster I

Linking DNA Damage Response to Ferroptosis for Leukemia Stem Cell Eradication

Novel Role of Mitochondrial Folate Metabolism in Cell Fate Decisions and Leukaemogenesis

Regulation of Metabolic Homeostasis By TRAF6 Contributes to the Leukemia Progression

Loss of Function MBD4 Mutation Impairs DNA Repair in Association with TET2 in Acute Myeloid Leukemia

PGK1 Represents a Therapeutic Target for Pediatric Acute Myeloid Leukemia Via Regulating c-Myc/SLC7A5/mTOR Pathway

Targeting EYA1 Activity in MLL- Rearranged Leukemia

BCR-ABL1 Dislocates NPM1, PU.1 and KLF1 into Cytoplasm to Thereby Skew Granulo-Monocytic and Impede Erythroid Differentiation

Uncovering the Role of CD59 As a Novel Therapeutic Target Against High-Risk Acute Myeloid Leukemia

Loss of SMARCA4 Leads to an Impaired Hematopoiesis in Mice

Ebf1 Is Involved in Primitive Myelopoiesis and Contributes in Leukemia Pathogenesis

Loss of CCAAT-Enhancer Binding Protein Delta Promotes Acute Myeloid Leukemia Cell Proliferation and Survival By Upregulating Cyclin D1 Expression

Nuclear Condensates Are a Therapeutic Vulnerability in NPM1-Mutant Acute Myeloid Leukemia

Age-Specific Induction of Mutant p53 Drives Clonal Hematopoiesis in Adult Mice Leading to Acute Myeloid Leukemia

Easyab Unravels Oncokinase Driven Regulation of Splicing Factors in AML

STAG2 loss Induces HSC Programs By Modulating Accessibility of AP-1 Bound Enhancers

Development of a Fully Immunocompetent Adoptive Transfer In Vivo Model of Npm1cA;Idh2R140Q;Flt3ITD AML

Isoform Specific Roles for the Acute Myeloid Leukemia Stem Cell Antigen CD97

Inherent Genome Instability Underlies Trisomy 21-Associated Myeloid Malignancies

Allele-Specific Expression of Leukemia Genes Is Associated with Pathogenicity in Poor Risk AML

ZBTB7A Loss Promotes Synthesis of Lipids and Ketone Bodies in Myeloid Leukemia

The Zymogen Form of Caspase-1 Is Required to Fine Tune Excessive Cell-Intrinsic Inflammation in Acute Myeloid Leukemia

Targeting BRD4 to Counteract Cytokine-Induced Expression of PD-L1 and PD-L2 on Vascular, Osteoblastic and Stromal Niche Cells

Defective Necroptosis Mediates Chemotherapy Resistance in AML